大脳半球障害の反対側Blink reflexに及ぼす影響について

Influence of Hemispheral Lesions to the Contralateral Blink Reflex

I．はじめに

　最近，三叉神経の電気刺激や角膜の機械的刺激または電気刺激により誘発されるblink reflex（trigemino-fa—cial reflex）における上位中枢の影響が注目されている．現在までに，大脳半球障害におけるこの反射の変化が検討され，大脳半球の反対側trigemino-facial reflexに及ぼす影響について，病巣と反対側の①spinal trigemi—nal complexへのfacilitatory effect（Kimura^1），On—gerboer de Visser^2）），②顔面神経核およびbulbar la—teral reticular formationへのfacilitatory effect（On—gerboer de Visser and Kuypers^3），Danger^4）），③pon—tine trigeminal centerへのfacilitatoryまたはinhi—bitory effect（Dehen et al^5），Darian-Smith^6））などがあげられているが，報告者の間で必ずしも意見の一致がみられているわけではない^7,8）．

Electrically elicited blink reflexes were investigated in 28 patients with unilateral hemispheral lesion. The reflex evoked by electrical stimulation of the supra-orbital nerve contralateral to the side of the hemi-spheral lesion was absent or depressed in 14 out of 28 cases.

The abnormal patterns of the blink reflexes were decided into four different types (Type AB, Type AD, Type EB, and Type AB-EB). The Type AB, recorded in 4 cases, consisted of bilateral abscence of late corm ponet (R2) when the supraorhital nerve on the non-lesion side was stimulated.