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近年,肺がプロスタグランディン(以下PGと略す)をはじめとする種々のvasoactive substanceの代謝をおこない,ガス交換をおこなう臓器であると同時に一種のmetabolic organであるとする考え方が有力となってきた。
また最近PGの炎症における役割が注目され,PGE1による体温上昇作用,血管透過性増加作用,leukotaxis促進作用,またPGE1, PGF2αによるcollagen産生作用などが明らかにされてきた1)。
The possibility that the prostaglandins can act as mediators in the inflammatory response has been suggested by a number of researchers.
The present investigation, therefore, was designed to study the distribution of prostaglandins of the E and F type in serum and pleural exudate from patients with pleuritis.
Eighteen patients with carcinomatous pleuritis, 3 with tuberculous pleuritis, 4 with pleuritis of unknown etiology were studied. Test samples were obtained before breakfast and kept at -80℃ until use.
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