脳脊髄炎によるゴナドトロピン分泌障害が原因と考えられた射精障害

SEMINAL EMISSION LOSS CAUSED BY HYPOGONADOTROPIC HYPOGONADISM FOLLOWING ACUTE ENCEPHALOMYELITIS: A CASE REPORT

　症例は25歳の男性。急性脳脊髄炎のため入院，神経学的後遺症なく治癒したが，退院後まもなく射精障害が出現した。内分泌検査により視床下部機能障害に伴ったゴナドトロピン分泌低下が原因の射精障害と診断した。ゴナドトロピン（HCG, HMG）投与では効果は不十分であったが，酢酸ゴナドレリン（LH-RH）投与では著明な効果が得られた。

A 25-year-old man with the past history of acute ence-phalomyelitis presented complaining of seminal emission loss. The hormonal examinations for hypothalamic-pitui-tary function disclosed hypogonadotropic hypogonadism. The administration of HCG and HMG, which increased the semen volume, failed to improve azoospermia. Four months after subcutaneous injection of LH-RH, 2.3ml of semen with a large amount of sperm was confirmed. Although the cases of hypogonadism and/or hypopituitarism following acute encephalomyelitis are rare, some are thought to be overlooked.