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A CASE STUDY OF CONDUCTION APHASIA Masami Saito 1 , Katsumasa Minami 1 , Muneyuki Tsukamoto 1 , Shigekazu Okamoto 1 1Department of Psychiatry and Neurology, Kansai Medical University pp.877-886
Published Date 1975/8/1
DOI https://doi.org/10.11477/mf.1406203756
  • Abstract
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The authors have studied a 41 years old right-handed male patient, who developed typical features of conduction aphasia after cerebral apoplexy, clinically over one year and anatomically after his desease for acute purulent mediastinitis.

The results are as follows :

1) After disappearence of transient disorders of verbal expression in acute stage, the patient pre-sented a pure form of conduction aphasia lasting for a longer period of time while he had never shown any disturbance of verbal reception and understanding. Thus, the authors has been con-vinced of that the conduction aphasia is not a sub-type or a temporary feature in convalescence of sensory aphasia.

2) Paraphasia and disturbance of repetition were the only and main symptoms in oral speech, but the latter was less predominant than the former. Word amnesia and grammatical disturbance have never been found. In written language, paralexia and paragraphia were persistently manifested throughout the course.

3) Literal paraphasia was much more frequent than varbal paraphasia both in spoken and in written languages. It was also noted that the paraphasia was considerably influenced by patient's attitude and reaction to his situation and circumst-ance.

4) As to repetition of sounds, words or phrases, the longer the syllables the more the disturbances, of which the features mainly consisted of para-phasia and partial omission and the incidences varied in accordance with the differences of tones and meanings of the given words, in contrast to the disorders of secondary articulation.

5) Any other agnosic or apraxic disability was not revealed by the systemic neuropsychologicalexaminations.

6) Based on the findings, it can be said that the conduction aphasia is exclusively due to an im-pairment of inner speech process, particularly that of expressive programming rather than otherwise.

7) In autopsy, it was confirmed that the lesion in left Insula Reili involved the entire medulla but neither its cortex nor the adjacent areas in the temporal and parietal lobes. Hence, the medullar lesion of the left insula must be responsible while its cortical and other lesions are not indispensable so far as the pure form of conduction aphasia is concerned.


Copyright © 1975, Igaku-Shoin Ltd. All rights reserved.

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電子版ISSN 2185-405X 印刷版ISSN 0006-8969 医学書院

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