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SEE-SAW NYSTAGMUS Susumu Ishikawa 1 , Shigetoshi Higaki 1 1The Second Department of Surgery, Hiroshima University Medical School pp.1701-1707
Published Date 1973/12/1
DOI https://doi.org/10.11477/mf.1406203439
  • Abstract
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See-saw nystagmus is a peculiar type of dis-sociated rotatory-vertical nystagmus in which the eyes rotate conjugately clockwise and then counter-clockwise. In addition to the rotatory movements, the intorting eye elevates while the opposite ex-torting eye falls, making the alternating opposed vertical movements like a see-saw. Since the des-cription of the original case of see-saw nystagmus (Maddox, 1913), less than 30 cases have been re-ported. Pathophysiology of this rare condition is controversial and several sites, such as ocular sys-tem, brain stem, diencephalon, combined lesions or multiple levels, have been suggested to produce this nystagmus.

We have had the opportunity to study an inter-esting patient. A 22-year-old woman was admitted to our department on January 8, 1970, having noted oscillopsia following cervical injury. Four years prior to admission she recognized a bitem-poral hemianopsia by herself. However, she had not consulted a physician because of lack of any other trouble.

General physical and hormonal examination was normal. Visual acuity was 0.2 in each eye but corrected to 1.0 by concave lens. Mild bilateral optic atrophy and a typical bitemporal hemianopsia were demonstrated. A classical see-saw nystagmus was apparent in all directions of gaze and most marked in upward gaze. The nystagmus was diminished on distant gaze and became marked on near gaze.

Caloric stimulation revealed a slight imbalance between the two vestibular systems. The second and the third cervical vertebrae were tender on percussion and the bilateral greater occipital nerves were painful on compression.

Neuroradiological examinations revealed a large suprasellar mass lesion. Right frontal craniotomy was performed and a suprasellar epidermoid tumor was encountered. Cheesy material was removed and portions of the thin capsule were excised. Macroscopic diagnosis was confirmed by histological examination.

Following surgery the nystagmus became in-conspicuous for several days but returned to the preoperative state. The nystagmus was diminished with either eye patched and also relieved in supine position on the patient. Cervical sympathetic block with 8 to 10 cc of Xylocaine inhibited tem-porarily the nystagmus. This procedure probably gave the effect on the both of the anterior and the posterior sympathetic systems. Cervical sympathetic block was repeated and CDP-choline, which activates the brain stem reticular formation, was administered. The nystagmus became grad-ually inconspicuous, and then disappeared com-pletely 2 years and 8 months after the surgery.Aat that time visual fields recovered to almost normal.

In this patient multiple factors, such as dience-phalic, ocular and cervical, seem to play each role in causing the see-saw nystagmus. As suggested by Ishijima, Sekino et al., a see-saw nystagmus may be produced by a disturbance of a delicatebalance in the supranuclear mechanisms controlling ocular movements in the brain stem, where various inputs from the cerebrum, the vestibular system, the cerebellum and the neck are integrated by a complicated multisynaptis network and output signals are sent to the ocular motor nuclei.


Copyright © 1973, Igaku-Shoin Ltd. All rights reserved.

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電子版ISSN 2185-405X 印刷版ISSN 0006-8969 医学書院

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