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要約 目的:医原性増殖硝子体網膜症で,残存硝子体皮質が病態に及ぼす影響の報告。症例:34か月間に硝子体手術を行った硝子体手術の既往がある増殖硝子体網膜症新分類grade C 27例27眼を対象とした。網膜表面に接着した均質の半透明または透明膜を残存皮質と定義し,その範囲,部位,網膜再剝離の原因になった網膜裂孔との関係を,トリアムシノロンアセトニド併用の硝子体手術で検討した。結果:残存皮質は24眼にあり,近傍のレーザー凝固斑,医原性裂孔,2次性黄斑円孔が開放していた。結論:硝子体手術の既往がある増殖硝子体網膜症では,残存硝子体皮質が裂孔の再開放の原因になる可能性がある。
Abstract. Purpose:To report the role of residual vitreous cortex in the pathogenesis of proliferative vitreoretinopathy(PVR)following previous vitreous surgery. Cases and Method:This study was made on 27 eyes of 27 cases who received vitreous surgery for grade C PVR after new classification,and had a history of vitreous surgery in the past. Residual cortex was defined as lucent or semilucent homogenous membrane adherent to the retinal surface. Location of residual cortex,its extent and relation to retinal break were evaluated during triamcinolone-assisted vitreous surgery. Findings:Residual cortex was present in 24 eyes. Retinal breaks,laser spots or macular hole became open in the vicinity of residual cortex. Conclusion:In PVR following previous vitreous surgery,residual vitreous cortex may have induced reopening of retinal hole or break.
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