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TRAUMATIC POSTERIOR FOSSA HEMATOMA Tomoyuki Hamasaki 1 , Tarumi Yamaki 1 , Eiji Yoshino 1 , Toshihiro Higuchi 1 , Yoshiharu Horikawa 1 , Kimiyoshi Hirakawa 2 1Department of Neurosurgery, Saisei-kai Shiga-ken Hospital 2Department of Neurosurgery, Kyoto Prefectural University of Medicine pp.1083-1090
Published Date 1987/11/1
DOI https://doi.org/10.11477/mf.1406206012
  • Abstract
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The traumatic posterior fossa hematoma was re-garded as relatively rare thing, but recently, as the result of the prevalence of CT scanners, the number of reported cases is increasing.

We report nine cases of traumatic posterior fossa hematoma. We divided into two categories : one was the acute epidural hematoma, the other was the acute subdural hematoma with cerebellar con-tusion. Five were cases of the acute epidural hematoma, three were cases of the acute subdural hematoma with cerebellar contusion and a case had both an epidural and a subdural hematoma. All the cases had struck the occipital region and had the occipital bone fracture. The prognosis of the five cases of the acute epidural hematoma was excellent, but that of the four cases of the acute subdural hematoma with cerebellar contusion was poor and they all died inspite of the removal of the hematoma executed in three cases.

We estimated that the hitting forth was extreme-ly strong in cases of the subdural hematoma with cerebellar contusion, and that the momentary de-formity of the occipital bone might injure the cerebellum directly. Once a hematoma was pro-duced in the posterior fossa, it oppresses the brainstem and causes the acute hydrocephalus, so the state of consciousness and respiration deterio-rate suddenly. In cases of the acute epidural hematoma, appropriate surgical intervention could save the patients and resulted in good outcome. But in some cases of the fulminant type acute epidural hematoma of the posterior fossa caused by tearing the sinuses, though we have not experi-enced, patients die before the diagnosis and treat-ment. In cases of the acute subdural hematoma with cerebellar contusion, as there existed the bone artifact in X-ray CT of the posterior fossa, subtle high or low density area of the early stage cerebellar contusion could not be detected. Only the finding of the cerebellar swelling was noted in early CT. In such a case, the posterior fossacisterns and the fourth ventricle were completely disappeared. Once the cerebellar swelling occurred, the suboccipital craniectomy, removal of the sub-dural hematoma and the intracerebellar hematoma were not sufficient for the decompression. This disappearance of the posterior fossa cisterns was the indicator of the primary or seccondary brain-stem injury and the poor prognosis.

We reviewed reported cases of the posterior fossa injury and discussed about the mechanism of the production of the cerebellar contusion, the pathophysiology, the diagnosis and treatment.


Copyright © 1987, Igaku-Shoin Ltd. All rights reserved.

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電子版ISSN 2185-405X 印刷版ISSN 0006-8969 医学書院

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