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Japanese

COMPUTED TOMOGRAPHY OF RUPTURED ARTERIOVENOUS MALFORMATIONS IN THE ACUTE STAGE Shinichiro Takahashi 1 , Makoto Sonobe 1 , Taisuke Otsuki 1 , Yasuko Kubota 1 , Hiroshi Kawakami 2 1Division of Neurosurgery Mito National Hospital 2Division of Neurology Mito National Hospital pp.555-559
Published Date 1981/6/1
DOI https://doi.org/10.11477/mf.1406204770
  • Abstract
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Eighteen patients with angiographically proved intracranial arteriovenous malformation (AVM) were studied by computed tomography (CT) and their clinical features were reviewed at the same time. The pathophysiology of ruptured AVM in the acute stage was discussed.

Seven of the 18 patients were performed CT scan within 7 days after the onset. Although all but one of the patients showed symptoms and signs suggestive of subarachnoid hemorrhage (SAH), intracerebral hematoma was demonstrated in all cases and two of them were accompanied by ven-tricular rupture. On the more, no high density lesion was demonstrated in cerebral cisterns by plain CT scan as seen in the acute stage of ruptured cerebral aneurysms.

Six patients were performed contrast study within 7 days after the onset and showed no en-hancement effect. On the contrary, nine of 10 patients performed contrast study more than 8 days after the onset showed enhancement effect.

It has been commonly postulated that subara-chnoid hemorrhage is the main pathophysiology of ruptured AVM. However, our study on CT scan of ruptured AVM demonstrated that intra-cerebral hematoma or ventricular hemorrhage is the chief underlying pathophysiology of ruptured AVM in the acute stage.


Copyright © 1981, Igaku-Shoin Ltd. All rights reserved.

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電子版ISSN 2185-405X 印刷版ISSN 0006-8969 医学書院

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