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Variant angina due to deficiency of intracellular magnesium by anorexia nervosa Kazuhiko Tanabe 1 , Kiyohito Noda 1 , Akira Masaka 1 , Chihiro Nakanishi 1 , Satoshi Arai 1 , Yoshimitsu Kishi 1 , Koichi Sotozono 1 , Manabu Kamegai 1 , Fumihiko Miyake 1 , Takehiko Mikawa 1 , Masahiro Murayama 1 , Jiro Sugai 1 1The Second Department of Internal Medicine, St. Marianna University School of Medicine pp.467-471
Published Date 1989/4/15
DOI https://doi.org/10.11477/mf.1404205466
  • Abstract
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A 51-year-old man who had a past history of gastric resection for medically uncontrollable gastric ulcer has loss of appetite that recurs periodically. And he has frequently presented spontaneous angina early in the morning since 1984. He was diagnosed as having variant angina by the documentation of typical ST elevation during anginal attack and also by showing coronary artery spasm (# 2 and # 12) during hyperventilation on coronary arteriography.

A large quantity of calcium blocking agents and nitrates could not improve his symptoms. Lack of intracellular magnesium by loss of appetite was suspected from a daily excretion of urine magnesium (5. 3 mEq) and magnesium tolerance test (56. 7%), To confirm the effect of magnesium administration, the second coronary arteriography was performed, After magnesium sulphate (80 mEq, hourly) was injected, coronary artery spasm could not be induced by ergonovine. And orally magnesium oxide, calcium blocking agents and nitrates were started. Anginal attack disappeared with increasing urine magnesium.


Copyright © 1989, Igaku-Shoin Ltd. All rights reserved.

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電子版ISSN 1882-1200 印刷版ISSN 0452-3458 医学書院

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