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Brodmann Areas 39 and 40: Human Parietal Association Area and Higher Cortical Function Yasuhisa Sakurai 1 1Department of Neurology, Mitsui Memorial Hospital Keyword: 角回 , 縁上回 , 言語性短期記憶 , 言語性作動記憶 , 頭頂葉性失書 , angular gyrus , supramarginal gyrus , verbal short-term memory , verbal working memory , parietal agraphia pp.461-469
Published Date 2017/4/1
DOI https://doi.org/10.11477/mf.1416200765
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Abstract

The anatomy and function of the angular gyrus (Brodmann Area 39) and supramarginal gyrus (Brodmann Area 40) are described here. Both gyri constitute the inferior part of the parietal lobe. Association fibers from the angular gyrus project to the dorsolateral prefrontal cortex via the superior longitudinal fasciculus (SLF) II/arcuate fasciculus (AF), whereas those from the supramarginal gyrus project to the ventrolateral prefrontal cortex via SLF III/AF. Damage to the left angular gyrus causes kanji agraphia (lexical agraphia) and mild anomia, whereas damage to the left supramarginal gyrus causes kana alexia (phonological dyslexia) and kana agraphia (phonological agraphia). Damage to either gyrus causes Gerstmann's syndrome (finger agnosia, left-right disorientation, agraphia and acalculia) and verbal short-term memory impairment. “Angular alexia with agraphia” results from damage to the middle occipital gyrus posterior to the angular gyrus. Alexia and agraphia, with lesions in the angular or supramarginal gyrus, are characterized by kana transposition errors in reading words, which suggests the impairment of sequential phonological processing.


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電子版ISSN 1344-8129 印刷版ISSN 1881-6096 医学書院

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