一過性に浅前房を生じた原田病患者の毛様体，脈絡膜剥離

Transient shallow anterior chamber in patients of Vogt-Koyanagi-Harada syndrome

　浅前房を呈した原田病患者2症例の前眼部を超音波生体顕微鏡（UBM）で検索し，通常の眼底検査では発見が困難な毛様体，脈絡膜剥離が両眼の全周に明瞭に観察された。この毛様体，脈絡膜剥離はステロイド治療によって軽快し，浅前房も改善した。1例では治療中に再発した浅前房が，毛様体，脈絡膜剥離の再発とともに生じていた。原田病の経過中に生ずる浅前房の成因は，炎症に起因した毛様体の浮腫によると考えられていたが，炎症によって生じた滲出液が上脈絡膜腔に貯留して毛様体，脈絡膜剥離となり，チン小帯が弛緩し，水晶体が膨化前進することが主因と考えた。

Shallow anterior chamber was present in two patients during the acute stage of Vogt-Koyanagi-Harada syndrome. Ultrasound biomicroscope, UBM, showed the bilateral presence of ciliochoroidal detachment involving the whole circumference. Topical and systemic corticosteroids resulted in disappear-ance of ciliochoroidal detachment and of shallow anterior chamber. Shallow anterior chamber and ciliochor-oidal detachment reappeared during recurrence of the disease in one case. Contrary to the accepted view that shallow anterior chamber in this syndrome is induced by inflammatory ciliary edema, the present findings seemed to show that accumulation of exudative fluid in the suprachoroidal space results in ciliochoroidal detachment, in relaxation of the zonules of Zinn and in anterior displacement of the swollen crystalline lens.