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ACUTE INTRACRANIAL HYPERTENSION Shinjiro Yamamoto 1 , Minoru Hayashi 1 , Tetsuro Yamamoto 1 1Department of Neurosurgery, School of Medicine University of Kanazawa pp.259-267
Published Date 1971/3/1
DOI https://doi.org/10.11477/mf.1406202869
  • Abstract
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In cases with acute intracranial hypertension following subarachnoid hemorrhage of rupturedaneurysm and acute traumatic subdural hemato-ma, the CSF pressure showed transient rises called "pressure waves". The waves superimposed on an elevated level of CSF pressure, and could be classified into two types.

One was slow waves in the range of 30 seconds to three minutes in duration, and the other being sharp waves of 15-30 seconds. It was particularly notable that while marked rises in the systemic arterial blood pressure occured synchronously with the sharp waves, opposite was true for the slow waves. The slow waves were usually observed in early or still not severe stages of the intrac-ranial hypertension while the sharp waves wereseen in advanced or maximal stages. When occurring continuously they appeared like rhyth-mic oscillations.

The pressure waves were susceptible to anes-thetics, particularly to barbiturates although they were somewhat depressed by steroids. Also sei-zure-like bursts in EEG appeared synchronouoly with the pressure waves. These suggest that the pressure waves are of neurogenic origin indicating an instability of the vasomotor mechanism of the cerebral vascular bed and an ominous sign of development of brain dysfunction.


Copyright © 1971, Igaku-Shoin Ltd. All rights reserved.

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電子版ISSN 2185-405X 印刷版ISSN 0006-8969 医学書院

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