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A Case Report of AA Amyloidosis associated with Cystic Infection during the Course of Non-tuberculous Mycobacteriosis Yukihiro Zaizen 1 , Kazuhiro Arita 2 , Hiroshi Kawano 3 , Taiki Watanabe 1 , Kazuhiko Hashinaga 4 , Tohru Yamazaki 5 , Junichi Kadota 4 1Department of Internal Medicine, Nishibeppu National Hospital 2Department of Internal Medicine, Oribe Hospital 3Department of Respiratory Medicine, Nishibeppu National Hospital 4Division of Pathogenesis and Disease Control, Department of Infectious Diseases, Oita University Faculty of Medicine 5Department of Respiratory and Blood Medicine, Oita Prefectural Hospital Keyword: AAアミロイドーシス , 非結核性抗酸菌症 , ジメチルスルホキシド , amyloidosis , non-tuberculous mycobacteriosis , dimethyl sulfoxide pp.237-241
Published Date 2007/2/15
DOI https://doi.org/10.11477/mf.1404100545
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Amyloidosis is a disorder affecting multiple organ systems and is caused by infiltration of tissues by amyloid. We describe the case of a patient in whom amyloidosis developed. The clinical course led to the suspicion of infectious lung cyst and non-tuberculous mycobacteriosis as the cause. The patient was a 73-year-old male who had been sent to this hospital because of his positive sputum for AFB(Acid Fast Bacilli). About two weeks prior to his visit here, the patient felt as if he had caught cold, because of fever and cough. He went to his family doctor and had a chest X-ray which showed an abnormal finding in the right lung field. The physician suspected pulmonary tuberculosis and examined his morning sputum for AFB. The sputum showed Gaffky 5 on smear, so he was referred to this hospital to evaluate his lung disease. On admission, the sputum showed Gaffky 4 on smear, and the AFB was identified as nontuberculous mycobacteria. A week after admission, he experienced a dull continuous pain in the abdomen. Weight loss and fever were also demonstrated and it was found that he had a history of anorexia A gastric fiber revealed diffuse depositions of amyloid in the gastrointestinal tract. This amyloid protein was identified as AA protein, suggestive of secondary amyloidosis. Pulmonary infection due to nontuberculous mycobacteria appears to have been the disease underlying this patient's amyloidosis. Amyloidosis was transitionally recovered, using dimethyl sulfoxide. However, amyloidosis took a turn for the worse and he died of amyloidosis five months after his admission to this hospital.


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電子版ISSN 1882-1200 印刷版ISSN 0452-3458 医学書院

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